Cytoplasmic male sterility is total or partial male sterility in hermaphrodite organisms, as the result of specific nuclear and mitochondrial interactions. [1] . Male sterility is the failure to produce functional anthers, pollen, or male gametes.

Cytoplasmic male sterility (CMS) is a maternally inherited trait that causes dysfunctions in pollen and anther development. CMS is caused by the interaction between nuclear and mitochondrial genomes.

Cytoplasmic Male Sterility (CMS) refers to the inability of a plant to produce viable pollens due to cytoplasm-based extranuclear genetic conditions under the control of the mitochondrial or plastid genomes. In general, there are two types of cytoplasm: N (normal) and aberrant S …

Cytoplasmic male sterility (CMS) is a maternally inherited loss of male fertility based on flower male organ dysfunctions that range from anthers in a low position or reduction in pollen fertility (the simplest cases) to a complete conversion of stamens into other floral organs.

Cytoplasmic male sterility (CMS), a condition under which a plant is unable to produce functional pollen, is widespread among higher plants. CMS systems represent a valuable tool in the production of hybrid seed in self-pollinating crop species, including maize, rice, cotton, and a number of vegetable crops.

The plant trait cytoplasmic male sterility (CMS) is determined by the mitochondrial genome and is associated with a pollen sterility phenotype that can be suppressed or counteracted by nuclear genes known as restorer-of-fertility genes.

Cytoplasmic male sterility is a maternally inherited genetic mechanism in higher plants that affects the production or functionality of pollen leading to male sterility. The genetic mechanisms causing CMS are very diverse in plants and vary even within the same species.

May 1, 1998 · Cytoplasmic male sterility (CMS) is a maternally inherited condition in which a plant is unable to produce functional pollen. It occurs in many plant species and is often associated with chimeric mitochondrial open reading frames.